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We speculate that LCS3 may be unique in its ability both to cause oxidative stress in NQO1-expressing cancer cells and to abrogate the ability of cancer cells to cope with accumulated ROS, through parallel disruption of the glutathione and thioredoxin systems

It has the capacity to promote angiogenesis, to regulate inflammation, and to induce more organized tissue repair

The concentrations of ROS, GSH, GSSG, NADPH and G6P are given by the model variables [ROS], [GSH], [GSSG], [NADPH] and [G6P], respectively

The expression or activity of GPX4 is influenced by glutathione (GSH), and GSH synthesis depends on the availability of cysteine [14]

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